Monoamine Oxidase Inhibitor vs Present Illness

In cardiac insufficiency with cardiac glycosides increase the contraction of the heart and make them more rare (eliminate tachycardia). With an overdose of the cardiac glycosides are also possible: nausea, vomiting (chemoreceptor stimulation triggerzony vomiting center), diarrhea, blurred vision, anxiety, psychotic reactions. Digoxin is used mainly in chronic heart failure, particularly in cases where chronic heart failure accompanied by atrial fibrillation. Na +, K + transport contributes ATFaza Na + ions from the cell and K + ions into the cell. Toxic garish of cardiac glycosides were more pronounced on the background hypokalemia and hypomagnesemia, as well as an increase in calcium levels. Cardiotonic properties are cardiac glycosides and 1adrenomimetiki. Vasodilating agents, expanding the garish vessels, reduce the blood pressure and thus reduce the workload on the heart. Sometimes used in acute heart failure; slowly injected glucose solution. Diuretics used in heart failure - hydrochlorothiazide furosemide, and others to increase excretion garish Na + and water, resulting in: decreases the volume of extracellular fluid (Reduces swelling), decreases the volume of blood plasma (reduces the load on the heart). In appointing the inside is not effective. Ouabain (Strophanthin) and Korglikon surpass the activity of digitalis drugs, act faster and shorter. Spironolactone - a weak diuretic, but the mechanism of action garish an antagonist of aldosterone and therefore is effective in chronic heart failure. Solutions of potassium chloride intravenously garish . When you receive systematic garish it possible material accumulation. Ca2 + ions bind to troponin C, which is included in the The complex troponintropomiozin. In acute heart failure intravenously administered high-vasodilator Tools - sodium nitroprusside, nitroglycerin. Less likely to use lanatozid C (Celanidum; predecessor digoxin), digitoxin (glycoside purple foxglove), ouabain (strophanthin; contains glycosides strophanthus) and Korglikon (contains glycosides lily of the valley). Celanidum acts slightly faster and weaker garish digoxin. The most frequently from use of cardiac glycosides digoxin, extracted from the woolly foxglove (Digitalis lanata). Cardiac glycosides difficult atrioventricular conduction and large doses can cause atrioventricular block. Increased automaticity of the Purkinje fibers explain decrease in the concentration of K + in the cytoplasm of cardiomyocytes. Different slow and long acting (t1 / 2 - 160 h). Angiotensin II: causes a narrowing of the arteries, stimulates the sympathetic nervous system (in particular, acts on the adrenergic nerve endings and increases the release of norepinephrine), stimulates the production of aldosterone, which leads to delay in the body Na + and water, increase in blood plasma. In chronic heart failure used isosorbide dinitrate, isosorbide mononitrate, similar to the action of nitroglycerin. For Oblique treatment of chronic heart failure is especially suitable carvedilol (dilatrend), who adrenoblokiruyuschee properties combined with a vasodilator (the blockade aadrenoretseptor) and antioxidant action. Blockers are traditionally regarded means contraindicated in heart failure, as these substances weaken the contraction of the heart. Apparently this is due to Mean Corpuscular Hemoglobin decrease in excessive sympathetic nervous system influences on the heart, as well as antianginal and antiarrhythmic action. When tahiaritmicheskoy atrial fibrillation digoxin normalizes ventricular contractions due to the inhibition of atrioventricular conduction. here a systematic treatment of chronic heart failure, ACE inhibitors are used - captopril, enalapril, lisinopril, and others. Drugs cardiac glycosides differ in the way of introduction, activity, speed and duration of action. The Anterior Cruciate Ligament effect of cardiac glycosides appears relatively often as a therapeutic breadth drugs is small.